The Medical and Surgical Practice of NaProTECHNOLOGY, 341-344, 2004
Chapter 28: Effects of Stress
Author affiliations
- Pope Paul VI Institute for the Study of Human Reproduction, Omaha, Nebraska. ROR
Abstract
Physiological and psychological stress disrupts hypothalamic GnRH pulsatility via CRH-cortisol pathways, producing downstream impairments in LH and FSH secretion that manifest as anovulation, delayed ovulation, follicular phase prolongation, or luteal phase deficiency -- all of which are documented cycle-by-cycle on the CrMS chart. NaProTECHNOLOGY addresses stress-induced HPO axis dysfunction by identifying the specific cycle-level disorder through charting and targeted hormone profiling, then applying cycle-appropriate ovulation induction and cooperative hormone support alongside correction of the underlying physical or psychological stressor.
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Cite this article
Hilgers, T. W. (2004). Chapter 28: Effects of Stress. *The Medical and Surgical Practice of NaProTECHNOLOGY*, 341-344.
Hilgers TW. Chapter 28: Effects of Stress. The Medical and Surgical Practice of NaProTECHNOLOGY. 2004:341-344.
Hilgers, T. W. "Chapter 28: Effects of Stress." *The Medical and Surgical Practice of NaProTECHNOLOGY*, 2004, pp. 341-344.